Be sure that you keep your cycle lengths as short as possible, especially if you are using the oral form. Not only does this minimize the risk of side effects, but it also helps to protect your liver. The higher your dose, the shorter your cycle should be. As an example, if you are taking 40mg of Turinabol 10mg Magnus Pharmaceuticals per day, you can extend your cycle out to as long as 12 weeks. On the other hand, if you are using the maximum recommended dose of 80mg per day, you should cut your cycle length to six or eight weeks at the most.
There is a big controversy on this matter. There are some sources, which tells that turinabol depot is detectable up to 18 months and oral turinabol is detectable up to 12 months in worst-case scenario. Some other people on the forums tell that 6-8 weeks in more than enough. However, we cannot understand where they all took such huge and unreliable numbers from. Doping tests rarely shows chlordehydromethyl-testosterone indeed and it was widely used in 60es and 70es so there is a good evidence that real detection times are much shorter. Most likely this mistake happened because athletes used some long-living substances like deca long with it. Or, maybe, these sources misrepresent turinabol with the same deca, we have seen some product descriptions, supposedly made for turanabol but after reading we understood they were just copypasted from deca descriptions, stupid, isnt’ it? Also, just as brain-storming: YES, it quickly leaves the body and not detectable in the urine tests, but probably modern blood tests can detect it for longer time, especially when using injectable turinabol.
Because the ultimate goal of a steroid cycle is to increase strength and muscle size, the associated spike in estrogen which accompanies steroids such as Testosterone is considered undesirable. In order to disassociate the two effects, two classes of drug are used. Medications such as Nolvadex or Clomid target the estrogen receptors. They make it more difficult for the estrogen to exert it’s influence within the body thus allowing the testosterone to act more freely. The second class is aromatase inhibitors such as Femara. They target the aromatase enzyme itself in order to prevent the production of estrogen in the first place. Sometimes, it’s not always clear which option you should go with or even what the differences are between the two. Lets clear that up a little.